Does FcγRIII (CD16) mediate modified C-reactive protein-induced endothelial cells dysfunction?
首发时间:2008-09-10
Abstract:Atherosclerosis and its complications, in particular acute coronary heart disease (ACHD) is the single leading cause of morbidity and mortality world wide. Among the clinically useful risk markers for development of ACHD, the classical acute-phase reactant C-reactive protein (CRP) has received considerable attention. Accumulating evidence indicates that CRP is not merely a corollary marker but rather an actual mediator/modulator of the inflammation that contributes to ACHD.
keywords: CD16, C-reactive protein, Atherosclerosis
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Does FcγRIII (CD16) mediate modified C-reactive protein-induced endothelial cells dysfunction?
摘要:Atherosclerosis and its complications, in particular acute coronary heart disease (ACHD) is the single leading cause of morbidity and mortality world wide. Among the clinically useful risk markers for development of ACHD, the classical acute-phase reactant C-reactive protein (CRP) has received considerable attention. Accumulating evidence indicates that CRP is not merely a corollary marker but rather an actual mediator/modulator of the inflammation that contributes to ACHD.
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Does FcγRIII (CD16) mediate modified C-reactive protein-induced endothelial cells dysfunction?
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