分析了太原市新沟垃圾填埋场渗滤液的水质特征，并以COD+作为主要指标探讨了垃圾中有机成分，含水率，以及c、H、O、N等元素含量对渗滤液水质的影响。在此基础上，利用植物检测系统考察了渗滤液对种子萌发和幼苗生长的毒性效应。结果表明。太原市新沟垃圾填埋场渗滤液有机污染物浓度高、水质随季节变化较大，垃圾中有机成分、含水率以及c、H、0、N等元素含量影响渗滤液水质，可能改变其污染特性。高浓度渗滤液可抑制植物萌发和幼苗的正常生长，且表现出对染毒浓度和处理时间的双重依赖性；但低浓度渗滤液对植物萌发和幼苗生长过程没有显著影响，甚至在一定浓度条件下可促进植物的生长发育。

通过对太原市不同季节、不同监测位点大气二氧化硫（SO2）浓度的监测，分析了太原市SO2污染状况，指出在太原市对SO2排放企业的治理和管理工作势在必行。

利用全细胞膜片钳技术研究了硫酸镁（Mgs04）对s02衍生物刺激大鼠海马神经元电压门控性钠通道的调节作用。结果表明，Mgs04 对SO2 衍生物增大大鼠海马神经元钠电流的效应具有抑制作用，此作用随着SO2 衍生物浓度的增加而减弱，随着Mgs04 浓度的增加而增强。不同时间加入MgsO4 均会显著抑制SO2衍生物增大钠电流的效应，说明Mgs04不仅对SO2衍生物增大钠电流的效应具有抑制作用，而凡可即时和延效性地抑制此损伤效应的发生。研究结果提示，硫酸镁对SO2 衍生物刺激大鼠海马神经元钠通道具有抑制作用，这可能是MgS04 对SO2 引发的中枢神经系统损伤具有防护和治疗作用的机制之一。图4参14

BACKGROUND: Sulfur dioxide(SO2) and its derivatives are recognized as the factors responsible for cellular chromosome cleavage and genotoxicity and capable of inducing oxidative injury and changes in enzyme activity. The toxicity of S02 and its' derivatives, from the perspective of cell membrane injuries and neurotoxic effects in particular, remains currently unclear. OBJECTIVE: To study the characteristics of depolarization-activated potassium current in rat hippocampal neurons, and observe the changes in the current in response to cell treatment with SO2 and its derivatives.

The cytogenetic damage induced by municipal landfill leachate was studied using chromosomal aberration (CA) in mouse bone marrow assay. Results show that leachate samples collected in different seasons decreased the mitotic index (MI), and caused significant increases of CA frequencies in treatment concentration (Chemical oxygen demand (COD) measured by the method of potassium dichromate oxidation, CODCr)-dependent manners. Compared with the negative control, reductions of the MI of 54 and 38% were detected for the highest leachate concentration (CODCr 320mg/L) in mouse bone marrow treated with both samples. The frequencies of CA increased significantly with increasing concentrations of sample 1 from CODCr 40 to 320mg/L, and from 80 to 320mg/L after exposure to sample 2. In addition, aseasonal difference of MI and CA frequencies induced by leachate was observed. The results confirm that leachate is a genotoxic agent in mammalian cells, and imply that exposure to leachate in aquatic environment may pose a potential genotoxic risk to mammals and humans. The results suggest that the CA in mouse bone marrow bioassay is efficient in genotoxicity studies of leachate on mammals, and that there appears to be a correlation between the genotoxicity in mammal system and the chemical measurement (CODCr) of leachate. The results also indicate that different discharge guidelines and environmental quality standards should be established for eachates discharged from landfills to aquatic environment in different seasons.